Genetic variants in ADAMTS13 as well as smoking are major determinants of plasma ADAMTS13 levels

Qianyi Ma, Paula M. Jacobi, Brian T. Emmer, Colin A. Kretz, Ayse Bilge Ozel, Beth McGee, Chava Kimchi-Sarfaty, David Ginsburg, Jun Z. Li and Karl C. Desch

Article Information

vol. 1 no. 15 1037-1046

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  • Submitted February 8, 2017
  • Accepted April 22, 2017
  • Published online June 19, 2017.

  • Qianyi Ma, 1Department of Human Genetics,
  • Paula M. Jacobi, 2Department of Pediatrics and Communicable Disease,
  • Brian T. Emmer, 3Department of Internal Medicine,
  • Colin A. Kretz, 4Howard Hughes Medical Institute, and
  • Ayse Bilge Ozel, 1Department of Human Genetics,
  • Beth McGee, 5Department of Computational Medicine and Bioinformatics, University of Michigan, Ann Arbor, MI;
  • Chava Kimchi-Sarfaty, 6Hemostasis Branch, Division of Plasma Protein Therapeutics, Office of Tissues and Advanced Therapies, Center for Biologics Evaluation and Research, US Food and Drug Administration, Silver Spring, MD; and
  • David Ginsburg, 1Department of Human Genetics,2Department of Pediatrics and Communicable Disease,3Department of Internal Medicine,4Howard Hughes Medical Institute, and
  • Jun Z. Li, 1Department of Human Genetics,7Thrombosis and Atherosclerosis Research Institute, Department of Medicine, McMaster University, Hamilton, ON, Canada
  • Karl C. Desch, 2Department of Pediatrics and Communicable Disease, Statistics

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